The Chest Pain Center provides effective classification treatment for patients with chest pain through multidisciplinary cooperation, based on rapid and accurate diagnosis, risk assessment and appropriate treatment, which not only improves the ability of early diagnosis and treatment of ACS (acute coronary syndrome), reduces the risk of acute myocardial infarction or avoids the occurrence of myocardial infarction, but also accurately screens out patients with pulmonary embolism, aortic dissection and low-risk ACS to reduce misdiagnosis, missed diagnosis and over-treatment, improve the clinical prognosis of patients.
Chest Pain Center Emergency Department Requirements
① The director of the emergency department is willing to undertake the task of building a chest pain center;
②The functional partition of the chest pain center was set up: including triage table, acute chest pain clinic, rescue room, acute chest pain observation room and other areas;
③ A flow chart guiding the rapid triage, rapid diagnosis and treatment of acute chest pain and standardized diagnosis and treatment of acute coronary syndrome has been established and has been implemented;
④ For patients with acute chest pain, the first ECG can be completed within 10 minutes after the first medical contact;
⑤ The project of rapid bedside detection of troponin and D-dimer was carried out.
WESAIL Chest Pain Center Solutions
> Hypersensitive cardiac troponin I( hs-cTnI)
hs-cTnI can specifically and sensitively respond to myocardial injury, which is an essential reagent item for chest pain center. The high sensitivity of high-sensitivity cardiac troponin has a strong discrimination ability for small myocardial injury. Early diagnosis of myocardial injury can detect cardiovascular risk earlier.
> N-terminal pro-brain natriuretic peptide (NT-proBNP)
NT-proBNP can reflect the heart function, is generally used for the diagnosis of symptomatic heart failure patients, and in the treatment of patients with heart failure can be evaluated by testing the efficacy and used as a prognostic assessment of patients with acute coronary syndrome.
> D-dimer (D-Dimer)
The D-Dimer is derived from plasmin solubilized cross-linked fibrin clots. As long as there is active thrombosis and fibrinolysis in the body's blood vessels, D-dimer will rise. The increase or positive is found in secondary hyperfibrinolysis, such as hypercoagulable state, disseminated intravascular coagulation, kidney disease, organ transplant rejection, thrombolytic therapy, etc. Because of bacteremia and other diseases, it is easy to cause abnormal coagulation, which leads to the increase of D-dimer, especially in the elderly and hospitalized patients.
In addition to the above items, the hs-cTnI of immune analyzer, Myoglobin,CK-MB,H-FABP,MPO,Lp-PLA2,NT-proBNP,ST2,cTnI/CK-MB/Myo,D-Dimer,PCT, hs-CRP CRP and other rapid diagnostic reagent items that can obtain results in 2-8 minutes provide an overall diagnostic solution for chest pain centers.